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Conclusion: Our results highlight distinct mitochondrial phenotypes in NAFLD iPSC-Heps, including an increased reliance on mitochondrial respiration, elevated endogenous FAO, and impaired utilization of exogenous fatty acids associated with enhanced ROS production, suggesting mitochondrial dysfunction when exposed to excess fatty acids
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High-resolution single-cell reference atlas profiled dysregulation in endometrial cells and inflammatory conditions in women with and without endometriosis (36, 37)
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